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Finally, DADs result from calcium-handling abnormalities, whereby spontaneous calcium-release events from the sarcoplasmic reticulum (SR) through so-called ryanodine receptor type-2 (RyR2) channels activate a transient-inward current, mediated largely by the sodium/calcium-exchanger (NCX), which depolarizes RMP and can trigger a new AP
DOI: 10.1007/s11095-005-2504-z Figure 12: Relevant protonation equilibria for haloperidol
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