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Psychiatry 13 , 898600 (2022)

Although initial studies suggested that GSH depletion was only a byproduct of oxidative stress generated during cell death, recent discoveries suggest that GSH depletion and post-translational modifications of proteins through glutathionylation are critical regulators of apoptosis

phosphate-buffered saline (PBS)] and gastric vein (+140 pM vs

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This process follows a known pathway in which the interaction between GLP-1 and its receptor, initiates Gs-protein coupling