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In the same study, it was determined that although GW9662 prevented the PPAR activation caused by rosiglitazone, it increased the anticancer effect of rosiglitazone

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Pike, L.S., et al., Inhibition of fatty acid oxidation by etomoxir impairs NADPH production and increases reactive oxygen species resulting in ATP depletion and cell death in human glioblastoma cells

AOD 9604 is a synthetic peptide derived from the C-terminal region of human growth hormone (GH fragment 176191), structurally modified with an N-terminal tyrosine residue to enhance stability in experimental systems