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Bauer G (2015) Increasing the endogenous NO level causes catalase inactivation and reactivation of intercellular apoptosis signaling specifically in tumor cells

While it seems that the introduction of GHRP-2 didn't result in any significant changes in the area covered by atherosclerotic plaques, it possibly led to a reduction in the production of superoxide in the aorta, as suggested by dihydroethidium staining

Hence, the exploration of new methods that offer a high cure rate, are cost-effective, and exhibit lower adverse reactions is extremely important

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Conversely, suppression of the SLC1A5 variant, a mitochondrial glutamine transporter, is sufficient to inhibit tumor growth by impairing glutamine metabolism in pancreatic cancer cells 16

2013;109 Suppl 2:S815