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In animal models, GLP-1RA therapy has been shown to slows atherosclerosis by inhibiting angiotensin-II-induced proliferation of vascular smooth muscle cells via AMPK activation [29]

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After 24 h incubation, cells were stained with organelle specific fluorescent probes by replacing the media content in each well with fresh media containing either 1 g/ml of Hoechst (nuclei tracker

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