[9] These proteins are members of the family of ATP-binding cassette transporters and can catalyse the ATP-dependent transport of a huge variety of hydrophobic anions, [10] and thus act to remove phase II products to the extracellular medium, where they may be further metabolised or excreted
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Importantly, neurological symptoms can occur even without anaemia and require prompt assessment
This mechanism is particularly relevant for conditions characterized by profound, unremitting fatigue
It provides reduced Glutathione, a powerful antioxidant
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