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The mechanisms underlying these effects include modulation of the skin immune response by suppressing the production of inflammatory cytokines, such as interleukin (IL)-1 and tumor necrosis factor-alpha (TNF), and increasing the production of anti-inflammatory cytokines, such as IL-4 and IL-10, therefore promoting a favorable environment for tissue healing and reducing inflammation (Routley and Ashcroft, 2009

doi: 10.1016/j.pnpbp.2023.110719 30 FanH.BaiQ.YangY.ShiX.duG.YanJ.et al

while CJC is longer lived

Physical Vitality

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Effect of DSS-induced ulcerative colitis and butyrate on the cytochrome P450 2A5: contribution of the microbiome