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Introduction Hepatocellular carcinoma (HCC) is the sixth most common cancer and the fourth leading cause of cancer-related death in the world (Williams et al., 2014)

Strategies such as combining copper ionophores with ferroptosis inducers, or superimposing DSF/Cu regimens on a background of GSH depletion or p53 activation, could overcome resistance to single-death modalities and deliver a potent ferroptosis-cuproptosis double punch to OSCC

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Additionally, upregulated Ras-RAF-MEK pathway expression may enhance cancer cell sensitivity to ferroptosis by modulating VDAC2/3 function and increasing ROS generation (40)

NAPQI is neutralized by glutathione (GSH)