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In a rat model of MCAO-induced stroke, neurogenesis was increased by semaglutide treatment, as evidenced by significantly increased levels of neurogenesis-specific biomarkers including nestin (intermediate filament protein that is a known marker of neuronal progenitor cells), doublecortin (microtubule-associated protein considered to be a reliable marker of neurogenesis), stromal cell-derived factor-1 (SDF-1) (chemokine promoting endogenous regeneration in the ischemic brain), and its receptor CXCR4

This helps explain why one small molecule can have such wide-ranging effects

In general, there is a relatively linear relation between the degree of prolactin elevation and the size of a true prolactinoma

May I ask how you know your deficiency was alcohol related and not autoimmune

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