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When the ROS generation rate exceeds the clearance rate, excessive oxidation of ROS might directly damage mitochondrial lipid membranes, proteins, and mtDNA, contributing to mitochondrial dysfunction

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Therefore, the mutant strain MG005 was constructed by interrupting the glgB gene in E

In contrast, another meta-analysis observed that the risk of fractures was reduced with liraglutide but increased with exenatide (also a GLP-1 receptor agonist, [63]

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