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This remains an active area of research [5]

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A recent study involving genetic regulation and pharmacological experiments in GLP-1R KO mice confirmed that GLP-1R signaling may be immunoregulatory, and that GLP-1R deficiency accelerates rejection reactions in a chronic heart transplant model induced by CTLA4-Ig (cytotoxic T lymphocyte-associated antigen-4) tolerance [16]

Measure baseline sex steroids and repeat testing after therapy to confirm meaningful shifts