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Stimulation of GLP-1Rs Elicits Cardioprotective Effects Through PI3K-Akt Axis Previous studies have been reported that exendin-4 ameliorated -cells death and enhanced mitochondrial biogenesis through Akt-dependent manner (Fan et al., 2010), and GLP-1 analog protected MG-induced apoptosis via EGFR/PI3K/Akt/mTOR pathway in PC12 cells (Kimura et al., 2013)

Fluctuations in blood pressure have also been noted as a potential side effect of BPC 157

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Both of these peptides can activate the receptor to the same maximal level as their corresponding full-length peptides (Figure 3a and b)