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In the cybrid cells, treatment or overexpression of MOTS-c was able to increase mitochondrial complex subunits (ubiquinol-cytochrome c reductase, complex III

PMID 18088327

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BPC-157 resists enzymatic breakdown which is precisely why oral delivery is a meaningful research variable for this compound, not a theoretical one.2 But that stability does not mean every delivery route produces the same result