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Excessive oxidative stress in mitochondria results in the release of abundant ROS, and the strength and magnitude of ROS signalling determines the effects of its downstream factors, which can induce the development of chronic immune disorders pathologically characterised by mitochondrial damage, including RA

Interestingly, research has indicated that the absence of receptor-interacting protein 140 significantly increases mitochondrial biogenesis and intracellular FAO [53]

Med 75 , 263275 (2020)

Results therefore highlight the importance of circadian regulation to fundamental cellular functions and support the hypothesis that disruption of core clock genes may lead to cancer development

Biomedicines 12 (2);405

Antioxidant defense mechanisms 4.1