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They inactivate MAP kinases by dephosphorylating threonine/tyrosine residues in the T-X-Y motif of the kinase activation loop 31

The oxidant stress that results from impaired homocysteine metabolism, which modifies the intracellular redox status, might play a central role in the molecular mechanisms underlying moderate hyperhomocysteinemia-mediated vascular disorders

doi: 10.1161/01.res.86.12.1252

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In our system, Cr GPDH further converts Gro3P to glycerol and Pi, which prevents Gro3P accumulation and allows uninterrupted NAD + recycling (because mGPDH is coenzyme Q linked, diminished electron flow via ETC decreases the activity of endogenous Gro3P shuttle and we previously observed elevated levels of both DHAP and Gro3P under conditions of NADH-reductive stress) ( Figure 1a ) 15