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Trx effectively prevents the progression of COPD by regulating redox status and protease/anti-protease balance, blocking the NF-B and MAPK signalling pathways, suppressing the activation and migration of inflammatory cells and the production of cytokines, inhibiting the synthesis and the activation of adhesion factors and growth factors, and controlling the cAMP-PKA and PI3K/Akt signalling pathways

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The activation of these pathways is initiated by different molecular signals depending on the etiology of AKI, as presented in Figure 2

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