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All data are expressed as the mean SD from at least three biological replicates, and comparisons between the two groups were performed with a nonparametric method (MannWhitney U test)

These modifications reduce affinity for IGF-binding proteins

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In patients with persistent or permanent AF but without pronounced LV dysfunction, the classical mechanism of atrial arrhythmogenesis is observed, characterized by reentry-promoting shortening of atrial APD [119, 120, 123]