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Notably, genetic studies have implicated microglial dysfunction in AD risk: rare variants in microglial genes such as TREM2 , which encodes an innate immune receptor on microglia, substantially increase AD risk, presumably by impairing microglial ability to respond to and clear amyloid

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Compared to saturated fatty acids, PUFAs are easily oxidized and accumulation of such PUFAs cause death of tumour cells through ferroptosis [104, 260]

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Kidney Int 98:14071418