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Consider the medications pharmacology
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The genes affected by semaglutide included markers of inflammation and plaque stability that are associated with leukocyte recruitment [e.g., interleukin-1 (IL-1) receptor antagonist, IL-6, monocyte chemoattractant protein (CCL2), OPN], leukocyte adhesion [selectin E, vascular cell adhesion molecule-1 (VCAM1)], leukocyte extravasation and plaque stability (e.g., MMP3, MMP13, CCL2), and plaque rupture and hemorrhage (CD163)
Micronutrient status was not measured systematically, so low reported rates of nutrition events do not establish nutritional adequacy
That is because we now know GLP-1 has an influence on all of these conditions, and that these conditions are associated with what we think is a generalised low-grade inflammation, which is harmful
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