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In animal models, MOTS-c treatment improved markers of mitochondrial health and reduced metabolic dysfunction associated with aging

It is thought to suppress the molecular expression of TNF- and other pro-inflammatory cytokines and inhibit the activation of the nuclear factor B (NF-B) pathway [65,66]

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PMID 8242618

Several studies have shown that specific promoters of genes, related to neural plasticity and learning and memory, could be modified by neuronal activity through methylation and demethylation [70]