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Back Samols, E, Marks, V (23 Feb 1963) 'Insulin assay in insulinomas', British Medical Journal , 1/5329, 507-10
The liver is exposed to: excess dietary sugars, especially fructose excess glucose from.digestion of carbohydrates consumed (poor muscle uptake) excess fatty acids (from insulin-resistant fat) It responds by converting this surplus into triglyceride and exporting it as VLDL particles effectively redistributing energy throughout the body
Early increases in bile acids post Roux-en-Y gastric bypass are driven by insulin-sensitizing, secondary bile acids
In the semaglutide group, 40% of patients exhibited increased residual gastric content, compared to only 3% of non-semaglutide users [18]