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Certain enteric pathogens possess mechanisms that enable their direct adherence to IECS while others permit the secretion of effector proteins or enterotoxins via membrane localised secretion systems that typically target TJs thereby increasing intestinal permeability by disrupting the intracellular cytoskeleton of IECs (Knig et al

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In phase 2, acetaldehyde is metabolised by ALDH to the inert metabolite acetic acid