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[1] These are exceedingly uncommon and possibly coincidental findings

466 One study proposed a novel mechanism wherein postprandial GLP-1 release, under normal physiological conditions, inhibits the activity of chemosensory cells in the carotid body, thereby counteracting sympathetic excitability mediated by elevated blood glucose or insulin levels

(1) The team discovered it while studying sleep-promoting factors in the brain and when they injected the isolated fraction into recipient animals, those animals showed a rapid increase in delta wave (slow-wave) sleep

For BPC-157 specifically, the authors noted favorable tissue repair findings in animal models while explicitly flagging scarce human safety data

6 This is not a trivial reformulation

Wakabayashi, H., Teraguchi, S., and Tamura, Y., Biosci