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GH-driven lipolysis raises free fatty acid flux to an already-stressed liver, a mechanism documented in hepatic steatosis research from Hepatology

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demonstrated that ferroptosis contributes to UC via ER stress-mediated IECs death

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YQF increased normal neuron counts, reduced inflammatory cytokines (TNF-, IL-2, IL-6), and elevated BDNF, TrkB, and NGF mRNA expression

In addition, puerarin inhibited intestinal epithelial barrier dysfunction by increasing the expression of TJs (ZO-1, occludin and claudin-1) [249, 250]