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Wir wissen heute auch, dass eine chronische Lithium-Therapie eine deutliche Abnahme der Aquaporin-2(AQP2)-Bildung an den Sammelrohren einleitet, und der durch Lithium ausgelste Diabetes insipidus renalis ist vermutlich, wenigstens teilweise, Folge dieser reduzierten AQP2-Bildung