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The discipline is progressing towards more individualised methodologies, emphasising the comprehension of genetic and physiological determinants that affect therapy results

NASH is a heterogeneous disease, and its phenotypic manifestation likely reflects the interactions of different primary drivers and coexisting disease modifiers

Biased agonism offers a way to tune the signal, the multi-organ distribution suggests indications still being explored, and the lesson of the antibody era, that a receptor claim is only as good as its validation, applies to every new tissue and target

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