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The whole process of radical-induced toxicity appears to stem from the generation of oxygen radicals (superoxide anion or singlet oxygen radical) from which other reactive oxygen species (ROS) and reactive nitrogen species (RNS) can be derived

According to the classic two-hits model, the increased insulin resistance, secondary to metabolic syndrome, determines an excessive lipid accumulation in healthy hepatocytes (first hit), mainly through an increased mobilization of free fatty acids from visceral adipose tissue to the liver

PC3 was cultured in Roswell Park Memorial Institute medium (Gibco, cat

In animal models, this preservation was associated with increased fat oxidation relative to storage

S.et al (2017a)