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Phase 3: metabolic adjuvancyovercoming homeostatic resistance The era of considering bariatric surgery and pharmacotherapy as competing options has ended

used exendin-4, an exendin-based GLP-1 analog, in hyperglycemia-induced bronchial hyperresponsiveness ex vivo, which resulted in bronchodilation that was inhibited by either cAMP-PKA antagonism or GLP-1 RA [90], a result consistent with other findings (Fig

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After incubation, the culture medium was removed, and cells were treated with varying concentrations of Lipo 20:0 BPD-PC (SM-102) and Lipo 20:0 BPD-PC (without SM-102)

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theoretical evidence for a two-step methyl cation transfer mechanism