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[22] Differentiating common forms of hyperthyroidism, such as Graves' disease, single thyroid adenoma, and toxic multinodular goiter is important to determine proper treatment

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The only published study applying this approach to date in autistic disorder found that the GSH:GSSG was significantly lower in whole cell extracts and mitochondria from lymphoblastoid cells obtained from children with autistic disorder compared to controls but there was no difference in response to nitrosative stress as measured by GSH:GSSG in mitochondria [80]

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Furthermore, the restored gut microbiota composition, particularly the enrichment of prevotella (a known butyrate producer), suggests a compelling mechanism for the ameliorated oxidative stress and potential enhancement of gut and BBB integrity [39]

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