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Inflammatory mechanisms may play a crucial role in BD pathophysiology via their regulation of synaptic transmission/plasticity and neuronal survival [67, 68]

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A recent small scale study in healthy humans aged 1985 measured the ratios of cysteine:cystine and GSH:GSSG in plasma [99]

More recently, Fisetin treatment in SARS-CoV-2 infection displayed a significant activity in vivo by eliminating senescent cells and reduced mortality associated with the virus 22