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We further demonstrate that oxidative mitochondrial signalling can be used as a biomarker of response to fluoropyrimidine-based chemotherapy treatment in patients with CRC and may have clinical utility for stratifying patients at higher risk who could benefit from metabolically targeted combination therapies

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Further complicating this pathway, it has been demonstrated that a metabolite of cysteine, hydrogen sulfide (H2S), can regulate CRH release effectively (Kimura 2002)

Since sermorelin supports growth hormone production and TRT addresses testosterone, the therapies target different hormone systems and can complement each other under proper medical management