For example, exposure of mouse AD model to either enriched environment or voluntary physical activity, or combination of both reversed astrocytic atrophy and decreased -amyloid load
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Increased cellular protein S -glutathionylation sensitized hepatocytes to TNF hepatotoxicity Increased cellular glutathionylated protein levels in response to GSSG accumulation prompted us to examine the effects of cellular protein S -glutathionylation on TNF-induced cell death
