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UA prevented the death of dopaminergic neurons caused by hydrogen peroxide (H 2 O 2 ) or 1-methyl-4-phenylpyridinium (MPP+) in a cellular model of PD [102, 168]

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studies on personalisation are now beginning to emerge, with a recent finding that genetic variations in the GLP-1 receptor, and also in arrestin B1, are important for the glycaemic effects of GLP-1 receptor agonists [105]