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In experimental models of lipopolysaccharide (LPS)-induced inflammation, which simulate conditions akin to bacterial infections, GPS has been demonstrated to suppress macrophage activation (Wang et al., 2019)

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Additionally, in vitro experiments showed that exosomal SELENOP potentially crossed the BBB and supplemented Se to neuronal cells (mouse neuroblastoma N2a cells), inducing the production of intracellular selenoproteins