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The chronic pain problem: where the evidence and the biology diverge The most common leap I see is the leap from maybe helpful for an acute tendon injury to maybe helpful for arthritis, chronic knee pain, or long-standing degenerative issues. This is where I think the case is weak

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Its mechanisms include: Actin polymerization mobilizes cellular building blocks for tissue repair Cell migration accelerates fibroblast and keratinocyte movement to injury sites Angiogenesis promotes new blood vessel formation via VEGF upregulation Anti-inflammatory reduces IL-1 and TNF- at injury sites TB-500's smaller molecular size gives it excellent tissue distribution throughout the body