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GLP-1-based therapies and resmetirom operate through distinct and potentially complementary mechanisms: while resmetirom exerts direct hepatic effects by activating THR- to enhance mitochondrial function, reduce hepatic lipogenesis, and promote lipid oxidation specifically in hepatocytes, GLP-1 RAs and multi-agonists address the upstream metabolic drivers of MASLD including insulin resistance, obesity, and systemic inflammation

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[1] [2] It is indicated for the treatment of type 2 diabetes mellitus in adults, typically as an adjunct to diet and exercise

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A personal or family history of certain thyroid tumors and a known allergy to Semaglutide are commonly cited contraindications

Prospectives and challenges of nano-tailored biomaterials-assisted biological molecules delivery for tissue engineering purposes