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Therefore, more research should explore the role TIMP-2 and MMP-2 may have in PTSD, and how they not only relate to central oxidative stress processes, but common somatic comorbidities in PTSD as well

Likewise, increased platelet activation and consumption due to tirzepatide-mediated endothelial effects, or GLP-1/GIP receptor interactions leading to megakaryocytic suppression, remain speculative without supporting evidence [19]

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Membrane-bound O-acyltransferase 1/2 (MBOAT1/2) is a ferroptosis suppressor (Liang et al., 2023)