Data show that up to two-thirds of patients with HFpEF have CAD or coronary microvascular dysfunction including ischaemia, which leads to disruption of LV diastolic and systolic function, development of fibrosis, increased myocardial stiffness and cardiac remodelling
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These results indicate that the Epitalon peptide effectively promotes the neurogenic differentiation of hGMSCs, supporting its potential application in regenerative medicine, particularly for neural tissue repair and neurodegenerative disease treatment [1]