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The surplus fatty acids, if left unprocessed, divert to alternative metabolic pathways, yielding highly reactive lipid peroxides

Nevertheless, evidence was obtained that the enzymatic reaction is also required to sustain ascorbate accumulation induced in these conditions (Hamada et al

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28 Excessive ROS production leads to an oxidative inactivation of anti-proteases, alveolar epithelium damage, increased neutrophil retention and increased expression of various inflammatory mediators in the pulmonary microcirculation, aggravating the development of COPD

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