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pylori infection induces GC cells to express L-plastin via CagA-activated mitogen-activated protein kinase 1 (ERK) signaling pathway to mediate SP1 binding to L-plastin promoter, which promotes proliferation and migration [44]

This neuronal circuit driven by GLP-1 released within the OB could thus be involved in the physiological control of insulin secretion both in the pre-prandial and post-prandial states

The compound was never approved as a pharmaceutical treatment for obesity because the Phase 2 clinical trial results, while showing safety and modest efficacy, did not meet the threshold for drug registration

However, this is up for debate

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