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A significantly increased level of NO was found in brains with SZ than those of normal and non-schizophrenic psychiatric controls ( PURINE CATABOLISM In addition to GSH redox coupling mechanism and NO signaling, purine catabolism ( Figure 2 ) may be a previously unappreciated component of the homeostatic response of mitochondria to oxidant stress and may play a critical role in slowing progressive mitochondrial dysfunction in certain disease states ( c oxidase is a key enzyme in the mitochondrial electron transport chain

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Bachmann, M

Nat Rev Nephrol 8 , 122128 (2012)