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Excessive production of TNF- triggers a decrease in nitric oxide (NO), thereby inducing an increase in endothelin1 (ET-1), causing the proliferation of vascular smooth muscle (Barale and Russo 2020)

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These data confirm the central role of GPX4 in the regulation of intracellular redox states and ferroptosis in Friedreichs Ataxia

Mitochondrial damage causes inflammation via cGAS-STING signaling in acute kidney injury

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