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Las altas concentraciones de homocistena pueden promover el desarrollo de aterosclerosis (aterognesis) y trombognesis a travs de mecanismos que involucran estrs oxidativo y disfuncin endotelial, inflamacin, coagulacin sangunea anormal, y metabolismo lipdico desordenado (revisado en 35)

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The peptide also promotes polarization of macrophages from pro-inflammatory M1 phenotypes toward tissue-repairing M2 phenotypes, with treated macrophages showing 70% increased phagocytic activity for clearing inflammatory debris and 3-fold higher production of VEGF compared to untreated controls