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The role of mitochondrial transition pore, and its modulation, in traumatic brain injury and delayed neurodegeneration after TBI

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With chronic stress, oxytocin is thought to have a longer term stress-antagonistic function, partially via cortisol-mediated negative feedback on CRH, partially via GABAergic inhibition of CRH neuron function and partially via a direct inhibitory effect of oxytocin on CRH expression (90)