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In health, postprandial glucose elevation triggers pancreatic beta-cell insulin secretion in a biphasic pattern, facilitating glucose uptake in skeletal muscle, adipose tissue, and hepatic suppression of gluconeogenesis

The danger hypothesis was first proposed in 1994 by Polly Matzinger as a substitute for the non-self-hypothesis, suggesting that in the absence of tissue damage and inflammation and therefore danger signals, an immune response will not be mounted

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Patients should work with their healthcare provider to establish a personalized treatment timeline, which may span months, years, or potentially indefinitely depending on their metabolic health trajectory and maintenance of results