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Since these pathogen proteins and metabolites are often similarly structured to ones created by the human host, such molecular mimicry makes it difficult for the host to recognise the foreign pathogen (193), resulting in the hosts immune system becoming increasingly sluggish and more susceptible to acquiring other infectious agents

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In particular, lower levels of adiponectin and increased expression of TNF- and its soluble receptor have been recently related to the development of NASH (43)

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