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GAA-FGF14 ataxia (SCA27B): phenotypic profile, natural history progression and 4-aminopyridine treatment response

Yet removing them does not repair dysregulated reward circuitry

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Furthermore, TLR4 involvement was probed in each experimental parameter due to its association with the amplification of placental oxidative stress and insufficiency in other placental pathologies [42,43,44,45]

[CrossRef] [Google Scholar] Screening gut microbial trimethylamine production by fast and cost-effective capillary electrophoresis