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This prolonged retention might increase intragastric pressure and potentially promote retrograde movement of gastric contents into the esophagus, particularly in patients with pre-existing lower esophageal sphincter dysfunction or hiatal hernia

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Mechanistic investigations revealed that H8 modulated lipid metabolism through dual mechanisms: inhibition of 11-HSD1 and activation of the AMP-activated protein kinase (AMPK) signaling pathway

Currently, it is documented that the major redox control for these enzymes is a feedback inhibition exerted by GSH on GCL ( A