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The proposed mechanism involves maintaining synaptic vesicle integrity, competing for receptor sites, and potentially substituting for depleted neurotransmitters

Similar content being viewed by others Introduction Type 2 diabetes (T2D) is the leading cause of end-stage kidney disease (ESKD) globally [1]

Blinding in the STEP 1 to STEP 4 trials may have been compromised somewhat, however, by the fact that the primary outcome is based on a readily measurable, objective measure (weight loss) that patients can self-monitor, and by the large imbalance in GI AEs, a well-known complication of GLP-1 agonists

The increase of plasma TMAO concentration was positively correlated with left ventricular diastolic dysfunction

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