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doi: 10.18632/oncotarget.23208 9 SalvadorJGutierrezGLlaveroMGargalloJEscaladaJLpezJ

Millea PJ

Phosphorylation of Nrf2 at Multiple Sites by MAP Kinases Has a Limited Contribution in Modulating the Nrf2-Dependent Antioxidant Response

This suggests that direct COP1-p53 recognition may require additional cofactors, adaptors, or specific cellular signal conditions such as glucose induce assembly of CRL4 COP1 for p53 degradation, as suggested in Su et al 29

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that is highly expressed in Hepa1-6 cells, successfully suppressed tumor growth, and triggered a strong GPC3-specific cytotoxic T lymphocyte (CTL) immune response